{"id":2096,"date":"2017-08-01T12:51:01","date_gmt":"2017-08-01T12:51:01","guid":{"rendered":"http:\/\/cytologystuff1.wpengine.com\/non-gyn-atlas\/respiratory-cytology-dr-jim-linders-bonus\/"},"modified":"2025-02-10T20:04:39","modified_gmt":"2025-02-10T20:04:39","slug":"respiratory-cytology-dr-jim-linders-bonus","status":"publish","type":"page","link":"https:\/\/stage.cytologystuff.com\/zh-hans\/non-gyn-atlas\/respiratory-cytology-dr-jim-linders-bonus\/","title":{"rendered":"Respiratory Cytology – Dr. Jim Linder’s Bonus"},"content":{"rendered":"

[vc_row 0=””][vc_column 0=”” offset=”vc_hidden-lg vc_hidden-md”][vc_raw_html 0=””]JTNDY2VudGVyJTNFJTNDYSUyMGNsYXNzJTNEJTIyc2hpZnRuYXYtdG9nZ2xlJTIwc2hpZnRuYXYtdG9nZ2xlLWJ1dHRvbiUyMiUyMGRhdGEtc2hpZnRuYXYtdGFyZ2V0JTNEJTIyc2hpZnRuYXYtbWFpbiUyMiUzRSUzQ2klMjBjbGFzcyUzRCUyMmZhJTIwZmEtYmFycyUyMiUzRSUzQyUyRmklM0UlMjBUYWJsZSUyMG9mJTIwQ29udGVudHMlMjAlM0MlMkZhJTNFJTNDJTJGY2VudGVyJTNF[\/vc_raw_html][\/vc_column][\/vc_row][vc_row][vc_column][vc_custom_heading text=”Respiratory Cytology – Dr. Jim Linder\u2019s Bonus” font_container=”tag:h1|text_align:center” use_theme_fonts=”yes”][\/vc_column][\/vc_row][vc_row][vc_column width=”2\/3″][vc_column_text]<\/a><\/p>\n

DR. JIM LINDER’S BONUS:
\nOTHER RESPIRATORY STUFF<\/p>\n

<\/a><\/p>\n

Reminder: You may click on any slide image
\nfor an enlarged view.<\/strong><\/p>\n

\n\"Image<\/a><\/p>\n
Figure 155<\/strong>
\nFerruginous Body
\nFibers of asbestos, or other minerals may be coated with ferroprotein creating a “ferruginous body”. This structure is partially engulfed by a macrophage.<\/div>\n<\/div>\n
Figure 155<\/strong>
\nFerruginous Body
\nFibers of asbestos, or other minerals may be coated with ferroprotein creating a “ferruginous body”. This structure is partially engulfed by a macrophage.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 156<\/strong>
\nFerruginous Body
\nFerruginous bodies due to asbestos fibers typically have a clear central core.<\/div>\n<\/div>\n
Figure 156<\/strong>
\nFerruginous Body
\nFerruginous bodies due to asbestos fibers typically have a clear central core.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 157<\/strong>
\nCandida<\/em> spp.
\nMultiple hyphal forms of Candida<\/em> spp. may be recovered in the respiratory samples of patients with oral candidiasis. (silver-based stain)<\/div>\n<\/div>\n
Figure 157<\/strong>
\nCandida<\/em> spp.
\nMultiple hyphal forms of Candida<\/em> spp. may be recovered in the respiratory samples of patients with oral candidiasis. (silver-based stain)<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 158<\/strong>
\nPneumocystis carinii<\/em>
\nThe cyst forms of Pneumocystis carinii<\/em> have a central dark zone when stained with silver-based stains.<\/div>\n<\/div>\n
Figure 158<\/strong>
\nPneumocystis carinii<\/em>
\nThe cyst forms of Pneumocystis carinii<\/em> have a central dark zone when stained with silver-based stains.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 159<\/strong>
\nAlternaria spp
\nThe macroconidia of Alternaria<\/em> spp. has both longitudinal and septate hyphae.<\/div>\n<\/div>\n
Figure 159<\/strong>
\nAlternaria<\/em> spp.
\nThe macroconidia of Alternaria<\/em> spp. has both longitudinal and septate hyphae.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 160<\/strong>
\nPollen
\nPollen granules have a thick refractile wall, multiple surface spikes, and are typically pigmented.<\/div>\n<\/div>\n
Figure 160<\/strong>
\nPollen
\nPollen granules have a thick refractile wall, multiple surface spikes, and are typically pigmented.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 161<\/strong>
\nMacrophages
\nIn cases of lipoid, or aspiration pneumonia the macrophages contain multiple complex lipid vacuoles.<\/div>\n<\/div>\n
Figure 161<\/strong>
\nMacrophages
\nIn cases of lipoid, or aspiration pneumonia the macrophages contain multiple complex lipid vacuoles.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 162<\/strong>
\nLipid
\nThe lipid in cases of lipoid pneumonia may be highlighted by oil-red O stain.<\/div>\n<\/div>\n
Figure 162<\/strong>
\nLipid
\nThe lipid in cases of lipoid pneumonia may be highlighted by oil-red O stain.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 163<\/strong>
\nMacrophages
\nPatients who sustain injury to the microvasculature of the lung develop pulmonary hemorrhage, manifested by hemosiderin-containing macrophages.<\/div>\n<\/div>\n
Figure 163<\/strong>
\nMacrophages
\nPatients who sustain injury to the microvasculature of the lung develop pulmonary hemorrhage, manifested by hemosiderin-containing macrophages.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 164<\/strong>
\nLymphocytosis
\nPatients with sarcoidosis typically have an interstitial lymphocytosis.<\/div>\n<\/div>\n
Figure 164<\/strong>
\nLymphocytosis
\nPatients with sarcoidosis typically have an interstitial lymphocytosis.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 165<\/strong>
\nEpithelial Cells
\nHighly reactive epithelial cells may be seen in patients with pulmonary aspergilloma.<\/div>\n<\/div>\n
Figure 165<\/strong>
\nEpithelial Cells
\nHighly reactive epithelial cells may be seen in patients with pulmonary aspergilloma.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 166<\/strong>
\nCryptococcus<\/em>
\nMultiple intracellular yeasts are seen within the cytoplasm of macrophages in Cryptococcus<\/em> infection.<\/div>\n<\/div>\n
Figure 166<\/strong>
\nCryptococcus<\/em>
\nMultiple intracellular yeasts are seen within the cytoplasm of macrophages in Cryptococcus<\/em> infection.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 167<\/strong>
\nCryptococcus neoformans<\/em>
\nThe capsule of Cryptococcus neoformans<\/em> may be seen with mucin based stains.<\/div>\n<\/div>\n
Figure 167<\/strong>
\nCryptococcus neoformans<\/em>
\nThe capsule of Cryptococcus neoformans<\/em> may be seen with mucin based stains.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 168<\/strong>
\nCiliocytophthoria
\nRespiratory viral infections may cause tufts of cilia to become detached from bronchial epithelial cells. This process is termed ciliocytophthoria.<\/div>\n<\/div>\n
Figure 168<\/strong>
\nCiliocytophthoria
\nRespiratory viral infections may cause tufts of cilia to become detached from bronchial epithelial cells. This process is termed ciliocytophthoria.<\/div>\n
<\/div>\n

\n\"Image<\/a><\/p>\n
Figure 169<\/strong>
\nHyperplasia
\nReserve cell hyperplasia is characterized by tight clusters of small reserve cells.<\/div>\n<\/div>\n
Figure 169<\/strong>
\nHyperplasia
\nReserve cell hyperplasia is characterized by tight clusters of small reserve cells.<\/div>\n<\/div>\n
<\/div>\n

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